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 George Perry

George Perry

Professor
University of Texas
USA

Biography

George Perry is dean of the College of Sciences and professor of biology at The University of Texas at San Antonio. Perry is recognized in the field of Alzheimers disease research particularly for his work on oxidative stress. Perry received his bachelors of arts degree in zoology with high honors from University of California, Santa Barbara. After graduation, he headed to Scripps Institution of Oceanography and obtained his Ph.D. in marine biology under David Epel in 1979. He then received a postdoctoral fellowship in the Department of Cell Biology in the laboratories of Drs. Bill Brinkley and Joseph Bryan at Baylor College of Medicine where he laid the foundation for his observations of abnormalities in cell structures. In 1982, Perry joined the faculty of Case Western Reserve University, where he currently holds an adjunct appointment. He is distinguished as one of the top Alzheimer’s disease researchers with over 900 publications, one of the top 100 most-cited scientists in neuroscience and behavior and one of the top 25 scientists in free radical research. Perry has been cited over 42,500 times (H=103) and is recognized as an ISI highly cited researcher. Perry is editor for numerous journals and is editor-in-chief for the Journal of Alzheimers Disease. He is a fellow of the American Association for the Advancement of Sciences and past-president of the American Association of Neuropathologists. He received the Distinguished Professional Mentor Award from the Society for the Advancement of Chicanos and Native American Scientists. Most recently he has been named a Foreign Correspondent Member of the Spanish Royal Academy of Sciences. Perrys research is primarily focused on how Alzheimer disease develops and the physiological consequences of the disease at a cellular level. He is currently working to determine the sequence of events leading to damage caused by and the source of increased oxygen radicals.

Research Interest

Microbial Oceanography, Cytopathology of Alzheimer's Diseasem, The mechanism for RNA-based redox metal binding, The consequences of RNA oxidation on protein synthesis rate and fidelity, The role of redox active metals in mediating prooxidant and antioxidant properties, The signal transduction pathways altered in Alzheimer disease that allow neurons to evade apoptosis The mechanism of phosphorylation control of oxidative damage to neurofilament proteins